有氧運動消耗脂肪，阻力訓練消耗醣類。這是常見的說法。
任何運動類型對脂肪消耗，有一種關鍵過程
Utilization of skeletal muscle triacylglycerol during postexercise recovery in h
umans
B Kiens et al. Am J Physiol. 1998 Aug.
https://journals.physiology.org/doi/full/10.1152/ajpendo.1998.275.2.E332?rfr_dat
=cr_pub++0pubmed&url_ver=Z39.88-2003&rfr_id=ori%3Arid%3Acrossref.org
Abstract
The utilization of muscle triacylglycerols was studied during and after prolonge
d bicycle ergometer exercise to exhaustion in eight healthy young men. Two days
before exercise and in the postexercise recovery period, subjects were fed a car
bohydrate-rich diet (65-70% of energy from carbohydrates). Exercise decreased mu
scle glycogen concentrations from 533 +/- 18 to 108 +/- 10 mmol/kg dry wt, where
as muscle triacylglycerol concentrations were unaffected (49 +/- 5 before vs. 49
+/- 8 mmol/kg dry wt after exercise). During the first 18 h after exercise, mus
cle glycogen concentrations were restored to 409 +/- 20 mmol/kg dry wt. In contr
ast, muscle triacylglycerol concentrations decreased (P < 0.05) to a nadir of 38
+/- 5 mmol/kg dry wt, and muscle lipoprotein lipase activity increased by 72% c
ompared with values before exercise. Pulmonary respiratory exchange ratio values
of 0.80-0.82 indicated a relatively high fractional lipid combustion despite th
e high carbohydrate intake. From 18 to 42 h of recovery, muscle glycogen synthes
is was slow and muscle triacylglycerol concentrations and lipoprotein lipase act
ivity were restored to the preexercise values. It is concluded that muscle triac
ylglycerol concentrations are not diminished during exhaustive glycogen-depletin
g exercise. However, in the postexercise recovery period, muscle glycogen resynt
hesis has high metabolic priority, resulting in postexercise lipid combustion de
spite a high carbohydrate intake. It is suggested that muscle triacylglycerols,
and probably very low density lipoprotein triacylglycerols, are important in pro
viding fuel for muscle metabolism in the postexercise recovery period.
雖然這是室內腳踏車的測試，不是典型的阻力訓練運動而是有氧運動的典型。
實驗過程，觀察到肌細胞肝醣量從533 降至 108mmol/kg，而肌細胞脂肪量在運動前後都維
持在49mmol/kg，沒變化。
之後觀察到訓練後18小時，肌肝醣補升到409mmol/kg。
也觀察到肌脂蛋白脂解酶活性提高到72%。明顯表示，恢復的能源是以消耗血液脂肪酸為主
。
無論有氧或是阻力訓練，恢復時，肌纖維代謝會恢復肌肝醣。為了減少葡萄糖消耗，這時就
會增加消耗脂肪酸做為身體能量來源。
任何的訓練都有效。